By A. Julianov
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Choubey, D. (2010). Aim2 deficiency stimulates the expression of IFN-inducible Ifi202, a lupus susceptibility murine gene within the Nba2 autoimmune susceptibility locus. Journal of Immunology, Vol. 185, pp. J. (2011). Aim2 deficiency in mice suppresses the expression of the inhibitory Fcgamma receptor (FcgammaRIIB) through the induction of the IFNinducible p202, a lupus susceptibility protein. Immunol, Vol. 186, No. 12, pp. R. (2009). Predominant modifier of extreme liver cancer susceptibility in C57BR/cdJ female mice localized to 6 Mb on chromosome 17.
A. (2008). An Imputed Genotype Resource for the Laboratory Mouse. Mamm. Genome, Vol. 19, pp. W. (2002). Molecular pathogenesis of human hepatocellular carcinoma. Nature Genetics, Vol. 31, No. 4, pp. L. (2000). Preparation of PCR-quality mouse genomic DNA with hot sodium hydroxide and tris (HotSHOT). Biotechniques, Vol. 29, pp. , & Gu, J. (2004). Two Variants of the Human Hepatocellular Carcinoma-Associated HCAP1 gene and Their Effect on the Growth of the Human Liver Cancer Cell Line Hep3B. Genes, Chromosomes & Cancer, Vol.
Hepatitis infection is believed to increase the risk of developing HCC by 20 fold and is the major etiological factor in more than 80% of HCC cases (Anzola, 2004). , 2010). The standard treatments for early stage HCC, such as surgical resection and liver transplantation, can cure certain population of patients. However, due to the asymptomatic nature of early HCC and lack of effective screening strategies, 80% of patients present with advanced HCC at the time of diagnosis (Thomas and Abbruzzese, 2005).
Liver Tumors by A. Julianov