By Sylvie Andrieu, Helen Warren
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Extra resources for Ruminant Formula for the Future: Nutrition or Pathology?: Elevating Performance and Health
Durand, D. Gruffat and Y. Chillard, 1998. Mechanisms of liver steatosis in early lactation cows: effects of hepatoprotector agents. In: Proceedings of The Cornell Nutrition Conference for Feed Manufacturers, pp. 27-35. , F. Beaudeau, S. Billon, A. Robert and P. Faverdin, 2003. Effects of health disorders on feed intake and milk production in dairy cows. Livestock Production Science 83:53-62. Z. P. R. Ghorbhani and W. Kautz, 2003. Effects of bacterial direct-fed microbials and yeast on site and extent of digestion, blood chemistry and subclinical rumen acidosis in feedlot cattle.
Mulligan, R. Alibrahim and L. O’Grady lactogenesis and gestation are increasing; (b) a decline in calcium status with the onset of lactation: (c) an increase in the production of reactive oxygen metabolites; (d) an altered rumen fermentation resulting in varying degrees of ruminal or metabolic acidosis, (e) an acutely negative energy balance in the earliest weeks of lactation; (f) a myriad of endocrine changes associated with the onset of parturition, lactogenesis and resumption of ovarian cyclicity.
Therefore, there is a complicated but well documented link between nutritional status and immune status in dairy cattle in the periparturient period. 2. Cascade-like sequences of periparturient cow pathologies Periparturient health disorders are rarely isolated events. In many cases, one primary periparturient health disorder will lead to a whole sequence of health and productivity distortions that involves different biological systems within the body and different periods of time in the production cycle of the cow.
Ruminant Formula for the Future: Nutrition or Pathology?: Elevating Performance and Health by Sylvie Andrieu, Helen Warren