By Richard C. Dart
The five Minute Toxicology seek advice is a competent speedy entry advisor to assessing and treating poisoned sufferers. Designed and organised for speedy session and interested in functional scientific concerns, the publication covers the complete variety of chemical compounds, medicinal drugs, normal compounds, adversarial interactions, and sufferer shows with toxicologic factors. every one bankruptcy is split into usual different types flagged by means of icons - fundamentals, prognosis, Signs/Symptoms, therapy, Follow-up , and Pitfalls . a complete part is dedicated to comparing sufferers with suspected yet unidentified poisoning. no less than skilled, board qualified, training emergency physicians and toxicologists have been excited by the enhancing of each bankruptcy
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Additional resources for The 5-Minute Toxicology Consult
Nontoxicologic Causes Nontoxic causes of increased anion gap acidosis include any cause of acid accumulation. The most common cause is lactic acid arising from anaerobic glycolysis (ischemic tissue, hypoxia, severe agitation or seizure). SIGNS AND SYMPTOMS Associated physical signs may help reveal the poison involved when they occur in the setting of increased anion gap metabolic acidosis. Vital Signs Any cause of hypotension or hyperthermia may cause anion gap acidosis from lactic acid. HEENT • Blindness or blurred vision may indicate methanol poisoning.
Other drugs, such as amphotericin and methotrexate, can cause renal dysfunction. Renal Parenchymal Disorders • ATN can be caused by: acyclovir, allopurinol, aminoglycosides, amphotericin, amyl nitrite, aniline, arsenic, arsine, barium, bismuth, borates, bromides, captopril, carbamazepine, cephaloridine, cephalothin, chlorates, chromium, cisplatin, cortinarius mushrooms, cyclosporine, dapsone, diquat, elemental mercury, fluorinated anesthetics, glycols, halogenated hydrocarbons, insect venoms, methylbromide, methotrexate, mithramycin, naphthalene, neuroleptics, paraquat, phenazopyridine, radiocontrast media, snake venom, sulfonamides, rifampin, trichloroethylene, tetrachloroethylene, yellow phosphorus, and heme pigments.
Pitfalls It is important to anticipate the need for this procedure and obtain consultation early because: • CPB requires the involvement of other specialists to initiate. • CPB is not useful if the patient has hepatic failure. EXCHANGE TRANSFUSION Mechanism of Action Serial phlebotomy and transfusion such that the patient’s blood is gradually replaced with banked blood. Drug Interactions This procedure eliminates substantial amounts of most therapeutic drugs. Indications Exchange transfusion is generally performed in cases of: • severe methemoglobinemia where the patient is unresponsive to methylene blue therapy.
The 5-Minute Toxicology Consult by Richard C. Dart